Whilst watching a documentary on Australia’s most venomous (it didn’t mention Daniel Andrews who has been succeeded by Jacinta Allan – no, not Jacinda Ardern of NZ!) I saw a segment where a King Brown snake took on another deadly snake called a Taipan. It could do this because it was immune to the Taipa venom.
Entering the rabbit hole, I thought I would check out how it was able to do this and whether it was possible t conflate a theory that the experimental C19 mRNA concoctions were in fact a biological weapon that drew its inspiration from venoms to create an entirely synthetic poison that could be aerosolized and “fired” at chosen population centres.
After all, they have come up with airborne, mutating, self-replicating “vaccines” right?
So maybe they used the same technology with the full “virus” to try and spread a virus I the same way they can use a “vaccine” with bits amputated from it?
Side note: that SubStack article also mentioned this – no more news on that.
“Degrading the Spike Protein: Neanthes japonica (Izuka)”
Friday Hope: Degrading the Spike Protein: Neanthes japonica (Izuka) (substack.com)
Which had this:
“The fibrinolytic enzyme purified from Neanthes japonica (NJ) was first labeled as NJF. As you will read, it has the ability to treat two major pathological conditions caused by the Spike Protein.
It dissolves fibrin and fibrinogen (clots) and offers neuroprotection from reperfusion, which my previous research has shown to be a major cause of injury by the Spike Protein. It also inhibits lipid peroxidation and increases endogenous antioxidant defense enzymes.”
Dissolves fibrin and fibrinogen (fibrous white blood clots?).
Ok, back down the rabbit hole.
An article from 2006 here – remember the conflation of snake venom with the artificially lab-created spike protein (venom).
Science: An antidote for all snake bites | New Scientist
“Broady began tackling the antivenin problem after Japanese and Brazilian scientists independently discovered a substance in the blood of a poisonous snake which neutralises its own venom. The substance was presumed to protect snakes from poisoning themselves. Broady, together with students Michael Thurn, Su Yen Lau and Peter Hains, isolated a protein in the blood serum of the tiger snake. The protein, which they called Notechis scutatus inhibitor or NSI, inhibits tiger snake toxin.
In the laboratory, the Australians tested the protein against the venom of six other snakes: the common brown snake, the forest cobra (Naja melanoleuca), Russell’s viper (Vipera russelli), the half moon viper (Bothrops alternatus), the Central American moccasin snake (Agkistrodon bilineatus) and the western diamondback rattlesnake (Crotalus atrox). NSI inhibited the venom of all “,
Interesting. Especially if the treatment can also neutralize the spike protein (protein),
Deeper into the rabbit hole!
From here:
Opossum protein neutralizes nearly all poisons, could have benefits for humans (yahoo.com)
We have “Lethal Toxin-Neutralizing Factor (LTNF)”
It seems that its not only King Brown snakes that have developed some sort of immunity from snake bite but opossums too! All a bit tricky as the immunity is territory and snake species specific.
“Opossums may someday provide an antidote to nearly all forms of poison, including everything from snakebites to ricin.”
“The Journal of Venomous Animals and Toxins has found that the American opossum produces a protein known as Lethal Toxin-Neutralizing Factor (LTNF). And as the Boing Boing blog points out, the LTNF protein is exactly what it sounds like, seeking out otherwise lethal poisons that have entered an opossum's body and neutralizing them.”
“Conflating away, I went deeper down the rabbit hole and found this (confounding) article that brings is back almost full circle.
A snake venom enzyme shows anti-SARS-CoV-2 activity in vitro (news-medical.net)
Which was commenting on a pre-print that has now been peer reviewed and is here:
“These results suggest that snake PLA2s, in particular dimeric ones, are promising candidates for the development of antiviral drugs that target lipid bilayers of the viral envelope and may be good tools to study the interaction of viruses with host cell membranes.”
And maybe the experimental C19 mRNA and viral vector concoctions as well???
At this point, I ran out of air below ground and had to return to the sunlight of my ignorance, haha.2natural” and man-made spike proteins should not be viewed as the equivalent of the snake venoms that prevent respiration and circulation (and start the snakes digestive process).
It seems to me that the mechanism of action of poisons is different from infections that the body’s immune system can fight in multiple ways.
As it is, we wait for the work of the brilliant minds seeking a mechanism of action to counter each of the SV40 promoter/enablers, coli, DNA. e-coli. bacterial endotoxins (which seem to me to have the, mechanisms of action as that used by the Komodo Dragon to kill its prey – “bite and wait for prey to drop dead” – DNA fragments, metals, glass shards and all that before there is a global instruction to aspirate the injections rather than injecting them into a vein like a heroin addict!
Onwards!!!
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Wow. Ain’t nature grand…fascinating and terrifying all at once.
Like the psychopathology of the inventors of the spike protein and its aptly named clot shot.
IIRC isn’t Ivermectin derived from land-based earthworms?
And now we have Neanthes Japonica, a marine earthworm. Hmmm.
AND it destroys acute leukemia cells via induced apoptosis, apart from its clot-busting and SP removal abilities. The possibilities seem endless.
Leukemia paper here:
https://www.sciencedirect.com/science/article/abs/pii/S0145212613003342
Thanks Peter for sharing your rabbit hole discoveries, your intuitive ‘what if’ thinking and those links - now off to do a bit more reading!
PS Love the allusion to the scourge of Melbourne too.